BCL2A1 and neoplasm: We found that the JFK-induced cell proliferation arrest could be owing to a reduction in the levels of H3K4Me3 modification at SUSD2 and CCND2 in A549 cells. BCL2A1 is well-known antiapoptosis protein that converts into a proapoptotic protein when bound to NuBCP-9 [37]. TMEM158 is a transmembrane protein that is upregulated in response to activation of the Ras pathway and has been proposed to be a tumor-suppressor gene and a target gene in the mutator pathway [38].