This is in line with previous findings that BCL2 expression is dependent upon REL and RELA [40] to promote resistance to programmed cell death and important pro-survival functions [7,41], while BCL2L1 (BCL-XL), another anti-apoptotic protein, was observed to be upregulated by NFκB as a critical link between inflammation and cancer [4] and tumor progression [41]. The gene discussed is RELA; the disease is cancer.