It is believed that unbalanced expression of glycosyltransferases involved in the glycosylation pathways, such as T synthase, β1,6-GlcNAc-transferase, sulfotransferase, and sialyltransferases, as well as altered availability of precursor monosaccharide ­molecules, is responsible for altered glycosylation patterns in cancer and thus are probably the main factors responsible for enhanced availability of TF disaccharide (15–19). Here, TF is linked to cancer.