COL1A2 and scleroderma: Interestingly, average skin thickness of bleomycin‐treated Ctnnb1fl/fl; Col1α2‐CreER mutant mice was comparable to that of nontreated wild‐type mice (Fig. 2D), suggesting β‐catenin ablation in dermal fibroblasts at least partially rescued the scleroderma phenotype induced by bleomycin.