Instead, it was shown that the tryptase mMCP6 (and not the chymase mMCP4) could cleave IL‐6, and that abrogation of this pathway in dipeptidyl peptidase I‐deficient mice could possibly explain their elevated levels of IL‐6 and improved survival in a model of septic peritonitis 13. Here, CMA1 is linked to septic peritonitis.