LILRA4 and neoplasm: As the interaction between BST‐2 and ILT7 suppress pDCs‐mediated IFN responses required for deterring tumor growth 151, 152, it is tempting to speculate that elevated BST‐2 in tumors 119, 131, 153 and engagement of ILT7 by BST‐2 may contribute to tumor tolerance and progression.