All of the cervical cancer cells subjected to the MK treatment grew much more slowly and expressed less Akt1/p-Akt1, more p21/p27, less p-Rb and fewer molecules (p-GSK3β, β-catenin, c-myc and cyclin D1) of the Wnt/β-catenin signaling pathway than the cells that were not subjected to MK treatment (Figure S2). Here, MYC is linked to cervical carcinoma.