AR and posterior cortical atrophy: DHT treatment resulted in the induction of endogenous miR-203 levels in AR-positive LNCaP, LNCaP-AR (parental LNCaP overexpressing wild-type AR) [40], and CWR22Rv1 (22Rv1, a castration-resistant PCa cell line) cells, thereby establishing a positive correlation between AR and miR-203 in PCa cells, while the AR antagonist MDV3100 reduced relative miR-203 expression in LNCaP and LNCaP-AR cells but not in 22Rv1 cells (Figure 3A).