C9orf72 and Alzheimer disease: The sparing of tactile body schema coding in the present patients further suggests that the brain network substrate for altered self-processing in AD may be differentiated from the cortico-thalamo-cerebellar network substrate previously proposed to underpin the body schema deficit exhibited by patients with frontotemporal dementia due to C9orf72 mutations (Mahoney et al., 2012; Downey et al., 2014).