Moreover, we examined cardiac expression of Gtl2-Dio3 miRNAs in the mdx mouse model of Duchenne Muscular Dystrophy (DMD) and the DyW mouse model of laminin-α2 (merosin) deficient congenital muscular dystrophy type 1A (MDC1A), degenerative skeletal muscle diseases that have associated cardiomyopathy. This evidence concerns the gene MEG3 and Duchenne muscular dystrophy.