GH1 and glomerulosclerosis: Although the formal pathogenesis of progressive glomerulosclerosis in GH‐transgenic mice has been well characterized, the exact mechanisms inducing stimulation of renal and glomerular growth, glomerular and podocyte hypertrophy, mesangial and endothelial hyperplasia, and the well‐known subsequent renal lesion patterns seen in GH‐transgenic mice are yet not fully understood.