Recent research by The Cancer Genome Atlas (TCGA), together with intensive research over the last three decades, has revealed a large number of putative oncogenes that are amplified or overexpressed in glioma [4–6], including c-Myc and Cdc20. C-Myc is a well-known transcription factor with roles in cell cycle progression, apoptosis, and longevity during development [7] and oncogenesis [8] and in stem cell self-renewal, differentiation, and metabolic reprogramming [9, 10]. Here, MYC is linked to glioma.