Further,miR-10b inhibits epithelial endometriotic cell invasiveness by targeting Syndecan-1 (SDC1)36, suggesting that NR_033688 may associate with disease migration, whereas, endometrial miR-200c influences many events during normal and disease progression, such as hormone response cellular transformation, inflammation, and angiogenesis, which indicates this lncRNA may be involved with hormone mediated endometriosis progression. This evidence concerns the gene SDC1 and endometriosis.