Another group described the role of the oncogenic lncRNA PCAT-1 in prostate cancer cell proliferation through interaction with cMyc. Using a luciferase assay, the authors found that PCAT-1 was able to increase cMyc expression by binding to the 3′-UTR of cMyc. The authors went on to show that PCAT-1-mediated proliferation is dependent on cMyc protein stabilization. The gene discussed is MYC; the disease is Familial prostate cancer.