We observed similar results for the α‐2,6 sialic acid modification (compared to human OX40L; 121Lys, P > 0.5; 57Pro, P < 0.005; 114Asn, P > 0.3; 90Asn, P < 0.005; Fig 5F), clearly showing that 90asparagine of human OX40L is indispensable for the receptor function in influenza infection. This evidence concerns the gene TNFSF4 and influenza.