As shown in Figure 4C, after infection, the cyp83a1-3 mutant accumulated significantly more camalexin than the wild-type, and this increase was suppressed by a mutation in PAD3. In addition, we found that the transcript levels of the camalexin synthesis genes PAD3 and CYP71A13 were higher in cyp83a1-3 than in the wild-type in the absence of pathogen (Figures 4D,E). The gene discussed is PADI3; the disease is infection.