VPS26A and Parkinson disease: Although VPS26A and VPS29 interact with VPS35 in the CSC in the retromer complex, only two rare nonsynonymous variants (VPS26A, p.K93E; and VPS29, p.N72H) were identified in 702 PD patients, suggesting that mutations in VPS26A and VPS29 are not a frequent cause of PD (Koschmidder et al., 2014; Shannon et al., 2014; Gustavsson et al., 2015).