CAMK2G and ischemic cardiomyopathy: To determine whether the TAB generated model of non-ischemic cardiomyopathy used here recapitulates these reported findings, we assayed CaMKII phosphorylated at residue 286 (P-CaMKII), oxidized CaMKII (Ox-CaMKII), and total CaMKII (T-CaMKII) by western blot in ventricular tissue (Fig 2A).