Actually, under normal (LF) conditions, SST-KO mice did not differ from WT mice in tumor incidence, latency, multiplicity or burden, despite the fact that some in vitro and in vivo studies have shown that SST analogs can exert inhibitory actions in several cancer types, including pituitary, neuroendocrine and breast tumors [5–7, 48]. The gene discussed is SST; the disease is cancer.