TNFRSF1A and infection: Indeed, absence of TNFR1 on T-cells did notcompromise the control of M. tuberculosis infection over 6 months, and didnot lead to exacerbation of lung inflammation, cytokine or chemokine release.Further, TNFR1 on myeloid cells was also more crucial than T cell TNFR1 response ina model of reactivation of M. tuberculosis infection after control bychemotherapy, where the infection flares in the context of an established adaptiveresponse.