tuberculosis infection, in contrast to mice deficient for TNFR1 onmyeloid cells or complete TNFR1 deficient mice that succumb to primary orflaring infection with highly inflamed lungs, indicating that TNFR1 signallingin T cells is dispensable to mount an effective control of M.tuberculosis infection. The gene discussed is TNFRSF1A; the disease is infection.