Cell infiltration, necrosis andoedema were strongly elevated in M-TNFR1 KO and TNFR1 KO mice at21–28 days after infection, with a slightly stronger phenotype inM-TNFR1 KO on day 21, similar to TNF KO mice (Fig. 3e).TNF KO mice progressed to further inflammation by day 28, while WT controlledinflammatory cell infiltration, with no necrosis nor oedema. Here, TNFRSF1A is linked to infection.