FH patients had higher concentrations of total MVs (p = 0.022), endothelial cell-derived MVs (EMVs) (p = 0.002), erythrocyte-derived MVs (ErytMVs) (p = 0.043), monocyte-derived MVs (MMVs) (p = 0.004), and tissue factor-positive MVs (TF+ MVs) (p = 0.006), when compared to healthy controls (Table 2). The gene discussed is F3; the disease is familial hyperaldosteronism.