EEF2K and neoplasm: Overall, FOXM1 promotes important biological processes in TNBC cells, including cell survival, proliferation, invasion, migration, and tumorigenesis through regulating multiple signaling pathways including eEF2K and inhibition of FOXM1/eEF2K axis significantly block these events and tumor growth of TNBC, indicating that FOXM1 is a critical driver of progression of breast cancer and other cancers, representing a potential therapeutic target [5, 9, 10, 11].