Rather, CTX injury provoked a ~2.5-fold increase in the number of terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL)+/Pax7+ cells in cKOs after CTX injury when compared with WT mice, but not after BaCl2-induced injury, indicating that CTX injury promotes the apoptosis of SCs lacking C/EBPβ (Figures 1j and k). This evidence concerns the gene CEBPB and cerebrotendinous xanthomatosis.