The expression of other chemokine receptors (CCR1, CCR2, CXCR3) and of chemokines (CXCL9, CXCL10, CCL2, CCL3, CCL4, and CCL5) is increased but is unable to compensate for the lack of CCR5, suggesting that the CCR5 role in the infection with neurotropic flavivirus is non-redundant and distinct from encephalitides of other etiology [32]. The gene discussed is CCL3; the disease is infection.