In type 1 diabetes (T1D) the pancreatic β-cells are destroyed by the immune system in a process involving the proinflammatory cytokines interleukin-1-β (IL-1β), interferon-γ (IFNγ), and tumor necrosis factor-α (TNFα) released from antigen-presenting cells and T-cells [1, 2]. Here, IL1B is linked to type 1 diabetes mellitus.