IL4 and pneumonia: These observations are well correlated with the exacerbated pathology found in liver and lung and consequently mortality in Msmeg-PE11-infected mice similar to those observed in a Balb/c mouse model of pulmonary tuberculosis infection where appearance of IL-4 in the lung lesions coincides temporally and spatially with the appearance of areas of pneumonia and necrosis, leading to rapid clinical deterioration and death60.