Rosario et al. [11] applied transcriptomic analysis to the KGN cells and another human GCT-derived cell line, COV434, which in contrast to the KGN cells neither expresses FOXL2 nor contains the p.Cys134Trp mutation, consistent with it having been derived from a juvenile GCT [3]. The gene discussed is FOXL2; the disease is granular cell tumor.