TRIM32 and infection: However, compared with AdshRNA-infected controls, AdshTRIM32 infection-mediated TRIM32 deficiency dramatically exaggerated AngII-induced cardiomyocyte hypertrophy, as was evident by up-regulation of cardiomyocyte surface area (2419±280 compared with 1734±88 μm2; P<0.05; Figures 2B and 2C).