Upregulation of p53 by miR-125a-5p inhibitors was paralleled by the activation of a subset of p53-induced miRNAs, like miR-192 and miR-194, and was associated with the inhibition of cell growth, migration, and induction in apoptosis only of MM cell lines carrying a wild-type p53 gene [30]. The gene discussed is TP53; the disease is Miyoshi myopathy.