Through analysis of the TCGA breast and lung cancer patient cohorts we found that DYNLL1 expression significantly and inversely correlated with the hypoxia-inducible p53-dependent group of genes (Pearson r = −0.09197, p = 0.0013 for breast and Pearson r = −0.2618, p < 0001 for lung cancers, respectively) (Fig. 4E), suggesting that hypoxia- and p53-dependent repression of the ATMIN target DYNLL1 occurs in human cancers. This evidence concerns the gene ATMIN and cancer.