Bieneman et al.[7] confirmed reactivity of human blood basophils to ligands of TLR2 but not to TLR4, whereas Watanabe et al.[8] showed IgG1 and IgG4 production by B cells mediated upon basophil activation by TLR2 and 4 ligands from patients with autoimmune pancreatitis. This evidence concerns the gene TLR2 and autoimmune pancreatitis.