The results showed that radioresistance of tumors generated by RKIP overexpression CNE2-IR cells was significantly lower than that of tumors generated by control CNE2-IR cells, whereas radioresistance of tumors generated by RKIP knockdown CNE2 cells was significantly higher than that of tumors generated by control CNE2 cells as demonstrated by tumor growth and weight measurement (Figure 4A). Here, PEBP1 is linked to neoplasm.