Interestingly, defective phagocyte oxidative burst is the hallmark of the inherited condition chronic granulomatous disease (CGD), for which prophylactic IFN-γ therapy can reduce the number of serious infections and hospitalisations.7 However, no study to date has examined the utility of IFN-γ in improving phagocyte oxidative burst in SAH. The gene discussed is IFNG; the disease is chronic granulomatous disease.