As shown in Table 1, FSGS patients presented higher suPAR levels compared to MCD patients (3.67 ± 0.17 vs. 2.03 ± 0.18 ng/ml; p < 0.05), but significant lower levels of the uPA (0.1 ± 0.02 vs. 0.44 ± 0.15 ng/ml; p < 0.05). This evidence concerns the gene PLAU and focal segmental glomerulosclerosis.