Meanwhile, a previous study demonstrated higher hepcidin levels in NAFLD subjects; nevertheless, the research found no correlation between hepcidin and histologic findings.8 It was demonstrated that body iron stores in NAFLD regulated hepcidin.40 Therefore, it seems reasonable to adjust for patients iron storage when evaluating hepcidin levels in NAFLD patients. The gene discussed is HAMP; the disease is metabolic dysfunction-associated steatotic liver disease.