Thus, it is reasonable to hypothesize that genes associated with dyslexia and migrational error in animal models, such as KIAA0319 (Platt et al., 2013) and DCDC2 (Meng et al., 2005), contribute to orbitofrontal and superior temporal sulcus findings in people with reading disability. The gene discussed is DCDC2; the disease is dyslexia.