NFKB1 and prostate carcinoma: Then, always in prostate cancer (LNCaP and PacMetUT1) cells, it was reported that exposure of cells to DHA attenuated H2O2-induced NF-κB transcriptional activity and diminished the expression of the downstream anti-apoptotic target survivin; this activity was specific, since it was not observed in normal human prostate (PrEC) cells [136].