Henoch–Schönlein purpura (HSP), nowadays called IgA vasculitis, is an immune complex vasculitis affecting small vessels with dominant IgA deposits.1 HSP is a systemic vasculitis commonly seen in children.1 HSP is characterized by nonthrombocytopenic purpura, abdominal pain and bleeding, arthritis, and renal involvement.1 The manifestations of renal involvement vary from asymptomatic hematuria and/or proteinuria to nephritic syndrome, nephrotic syndrome, and rapidly progressive glomerulonephritis.2 The long-term outcome of HSP depends primarily on the extent of renal manifestations.2–4. This evidence concerns the gene CD79A and Henoch-Schoenlein purpura.