The literature is likewise divided over the role of ATM kinase activity during EBV infection; one report [42] concludes that ATM is important for efficient EBV lytic replication in Akata BX1 latent cells but another report [43] indicates that ATM is dispensable for EBV lytic replication in Tet-BZLF1/B95-8 cells. The gene discussed is ATM; the disease is Epstein-Barr virus infection.