In fact, the knock-down of CPT1A nuclear expression was sufficient to induce apoptosis and proliferative inhibition in MCF7, SK-BR-3 and MDA-MB-231 breast cancer cell lines, suggesting that the role of CPT1Av2, expressed only in cancer cells, is independent by the breast cancer phenotype molecular portraits, proposing a potential value as new tumor specific target. The gene discussed is CPT1A; the disease is neoplasm.