In addition, it has also been shown that a few well-known oncogenes, including H19, HOTTIP, Nespas, Kcnq1ot1, lincRNA-p21, mHOTAIR, Malat1, SRA, Foxn2-as, Gtl2-as, and H19-as, are involved in vitamin D receptor protection against skin cancer formation by maintaining the balance of oncogenic to tumor-suppressing lncRNAs [11]. The gene discussed is H19; the disease is skin neoplasm.