To address a causal role for lysyl oxidases in blunted alveolarization in the hyperoxia BPD model, newborn mice were treated with the pan-lysyl oxidase inhibitor β-aminopropionitrile (BAPN), which did not improve lung alveolarization, but did improve elastin organization assessed by visual inspection (89). The gene discussed is LOX; the disease is bronchopulmonary dysplasia.