Previous studies have demonstrated the regulation of BCL2 via the HH pathway through GLI1,40, 41 while other studies have reported BCL2 activation through GLI2.30 Bar et al.42 reported that HH/GLI-activated BCL2 was important for the survival of medulloblastoma (a cancer not examined in the present study). This evidence concerns the gene GLI1 and medulloblastoma.