Furthermore, since PRIMA-1Met suppressed both Mcl-1 and c-Myc leading to apoptosis in MM cells in our system (Figure 7), it is speculated that the two proteins could also interact in response to PRIMA-1Met building a network with miRNA-29a to control PRIMA-1Met-induced cell death in MM. The gene discussed is MCL1; the disease is Miyoshi myopathy.