The liver plays a central role in production and catabolism of Hcy and there is some data that Hcy is higher in patients with NAFLD, [65] but, in our study, we found no correlation between putative markers of NAFLD (ALT, GGT, TG, BMI) and Hcy (neither in absolute values before white wine drinking, nor comparing changes of these markers before and after wine consumption period; data not shown). The gene discussed is GPT; the disease is metabolic dysfunction-associated steatotic liver disease.