To investigate the possibility that OASL1 deficiency affects the trafficking of innate immune cells to the site of infection, we examined the proportion and the number of effector immune cell subsets, including pDCs, DCs, Ly6Chigh monocytes, neutrophils, and NK cells, in draining lymph nodes and vaginal tissues early after infection (Fig. 5). The gene discussed is OASL; the disease is infection.