POU5F1 and myocardial infarction: Neovascularization was significantly increased in MI hearts receiving c-kit+ CSCs, but the enhanced angiogenesis and atherogenesis were specifically inhibited in Oct3/4 siRNA-treated CSCs, suggesting that Oct3/4-mediated inhibition in microvessels in CSC-engrafted hearts might also be responsible for the improvement of myocardial function and the reduction of myocardial remodeling.