Thus, SOD1 was reported to reduce inflammation [38], accelerate the healing of skin lesions caused by burns, systemic lupus erythematosus, and herpes [39–41], protect cultured human neurons under oxidative stress [42], reduce ischemia-reperfusion injury [22, 43, 44], inhibit angiotensin II (AngII) intraneuronal signaling [19], prolong viability of β-cells [45], be effective in the treatment of rat adjuvant arthritis [46], and so forth. Here, SOD1 is linked to systemic lupus erythematosus.