Among these, consistent findings have been reported for ATOH7 [66,67,68,69], CDKN2B(-AS1) [66,67,69,70,74], GSTT1/GSTM1 [77,78,105,106], SIX1/SIX6 [69,70] and TMCO1 [92,93] loci, indicating a potential role of these genes/loci in the pathogenesis of POAG. Here, SIX1 is linked to open-angle glaucoma.