Indeed, induction of CDK1 has been linked to the growth of human colorectal cancer and acute myeloid leukemia [33–34]; the reduction of p27 has been liked to the genesis and progression of a broad array of human cancers, including cancers of the breast, colon, prostate, ovary, lung, stomach, and others [35–37]. Here, CDKN1B is linked to colorectal cancer.